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简介：Microfibrilledcellulose(MFC)isanewkindofnanoscalecellulosefunctionalmaterials,havingbroadapplicationprospectsinmanyfieldslikefood,medicine,cosmetic,paint,paperandpulpengineering,compositematerials,etal.TheresearchonMFChasbeenextensivelyconductedforthepast30yearsinsomedevelopedcountries,andevensomerelevantproductshaveappearedinthemarket.Onthecontrast,similarstudiesinChinawereratherlimited.ThispaperfirstlyintroducedthepropertiesandapplicationofMFCbriefly,andthengavemoredetaileddescriptiononthepreparationtechnologyofMFCanditsimportantperformanceindexes.SeveralsuggestionsonthefuturestudyonMFCwerealsoproposed.

简介：ObjectivesToanalyzethechangesofvasoactivesubstancesoriginatedfromendotheliuminpatientswithunstableanginapectoristreatedbymodifiedthrombolytictherapyandexplorethemechanismsofthedrugtotreatunstableanginapectoris.Methods120patientswithunstableanginapectoriswhowerenotwellresponsedtocommonmedicationwerestudied.TheirECGstresstestswereabnormalandtherewereischemicchangesinHolter.Urokinase300,000Uwasaddedin100mlnormalsalineandinjectedwithin30minonceadayfor3days.300mgaspirinwasadministratedadaybeforeandduringurokinaseapplications.Beforeandafterurokinasetreatments,endothelin-1,plasmatissueplasminogenactivatoranditsinhibitor-1weredetermined.ResultsComparedwithpretreatments,aftertreatments,theactivitiesoftissueplasminogenactivatorincreased,endothelin-1andtheinhibitor-1decreased.Thechangesweresignificant.ConclusionsModifiedthrombolytictherapycanregulatethevasoactivesubstancesoriginatedendotheliuminpatientswithunstableanginapectoris.Themajorsubstancesincludeendothelin-1,plasmatissueplasminogenactivatorandinhibitor-1.Thismechanismmaysuggestthaturokinasecantreatcoronaryheartdiseaseeffectively.

简介：<正>AttheinvitationofIndonesiaNationalCouncilonSocialWelfare(DNIKS)andSriLanka-ChinaFriendshipAssociation(SLCFA),Mr.LiuKaiyang,DeputySecretary-GeneralofCAFIUheadeda6-memberdelegationtovisitIndonesiaandSriLankafromDecember7to14,2014.Throughthisvisit,CAFIUhasenhancedmutualunderstandingwith